What is a power of Clomid?

Virtually every description of anabolic agents includes the slogan of unlocking and using clomid.
On our store’s website you will find a reliable and proven agent with a concentration of 50 mg/tab.
Clomid is a valuable agent for male athletes because it stimulates the secretion of hormones that affect endogenous testosterone secretion.
To summarize, after the cycle, it brings our economy back to normal without big drops, etc.
Clomid also reduces estrogen levels in the body, which prevents gynecomastia.
As for safety, it is a safe drug, as for dosage, with short cycles they say we take it from 4 to 6 weeks at 50 to 100 mg per day, with long cycles the first day is 250 mg, the next 3 days at 100 mg and until the end of treatment at 50 mg.
Remember, we start unblocking with HCG and only then do we start clomid.


Clomid is the trade name for Clomiphane Citrate. The compound is produced artificially in the laboratory in order to improve ovulation in women. But let’s not hide the fact that such a compound can cause additional effects, such as when men take it. Modern research confirms that clomid has a significant effect on maintaining testosterone production when it stops for natural reasons. Unfortunately, most sex steroid hormones cause a decrease in testosterone production in men. It is not surprising then, that after a steroid cycle, the body produces little or no testosterone of its own, and therefore unpleasant effects such as loss of muscle mass or gynecomastia can occur.
It is well known that testosterone causes a significant increase in muscle tissue in men.

The anabolic effect (increase in muscle mass) has been used for therapy. Under the watchful eye of chemists and in specialized laboratories it was possible to synthesize products with a structure very similar to that of male testosterone. It is worth knowing, however, that unlike the aforementioned hormone, they do not cause any androgenic effects, but have an anabolic effect: they increase the production of muscle-building protein, accelerate its construction, and increase the number of red blood cells, thanks to which the so-called “soreness” has no right to exist. The workouts are strong and painless, and the muscles “grow in your eyes.” Modern science is striving to achieve such an effect, in which a testosterone derivative does not upset the hormonal balance in any way, but only causes an anabolic effect. Despite numerous attempts, such an effect has not been achieved. There is always another bottom line. Even if the problem of hormonal imbalance does not occur while taking the steroid, it usually appears soon after stopping the drug. This is because that is when the body is most deficient in testosterone.

The problem is that only testosterone derivatives act on the appropriate receptors and cannot be replaced by anything else. If this hormone is delivered artificially, the body adapts to the new situation and stops producing it on its own or produces less of it. After therapy, the body switches to a different mode of operation. It is necessary to keep the muscles in good condition, so the oxygenation increases, the production of red blood cells increases, a different diet is required and more compounds are needed to keep the overgrown muscle fibers alive. The body, following the path of economy and new adaptation, does not “think” about the proper production of testosterone, hence the unpleasant effect – its deficiency, which gives the male body typical female features.


Clomid works positively on the hypothalamus so that it motivates the corresponding centers of the brain to produce gonadotropic hormones. These are folliculotropin (FSH) and luteinizing hormone (LH), which is also known as Intercellular Cell Stimulating Hormone. Folliculotropin stimulates the testes to synthesize testosterone, while LH causes testosterone secretion. This is a common hypothalamic-pituitary feedback mechanism that causes an increase in testosterone production and its release into the bloodstream. It is a natural compensatory system, working on the principle of equalizing concentrations.

The point is that clomid increases testosterone production and secretion to increase its level in the bloodstream, which should compensate for the decrease in exogenous hormones. This is the point of the steroid cycle – some kind of steady state, i.e. constant levels of certain hormones, should be maintained. Even a small change can disrupt their uninterrupted secretion. This is extremely important for maintaining the effect of muscle mass.
It is worth knowing that there are many more reasons to take clomid. The price of the product may not be low, but it gives very positive results. Namely, the agent acts as an anti-estrogen.

For a long time, it has not been known that estrogen is a typically female hormone responsible for certain female characteristics that can also manifest in men. Because clomid is a weak derivative of female estrogen, it demonstrates the ability to bind to estrogen-specific receptors. In contrast, it shows no ability to interact because, due to its extra molecular chain, it is unable to penetrate the receptor, which acts like a gate-it lets only the chemicals that are placed inside it through. An additional element in the conformational structure of the estrogen derivative forces the chemical compound to combine with one of the components of such a gate. However, due to the fact that it partially enters it, and hooks only one element, the gate is blocked for a longer time and becomes inaccessible to real estrogens, which could cause such active effects as gynecomastia.


It is good to know that the dosage should be adjusted to the steroid you are taking. Different steroid compounds have different half-lives because they are metabolized at different frequencies, rates and times. If it is taken when blood levels of androgens are high enough, the dose will have no effect.

On the other hand, if it is taken too late, the gains may be lost.
The half-life of Clomid is about 5 days. It is best to take a dose of 50-100 mg for 4-6 weeks in short cycles, and in long cycles take 250 mg for the first day, then 100 mg for 3 days, and finally 50 mg for 3 days and until the end of treatment.

The estrogen derivative is a substance that should carry out the correct dosage. Clomid should be taken in a 5:2:1 ratio, because only then can proper steady state be maintained in the bloodstream and normalize the release of your own hormones. The above dose is safe. The 50 mg tablets make dosing easy – there is no need to split or crush them.

Causes of female infertility

Infertility is suspected when a couple has not become pregnant during one year of active attempts. The causes of female infertility become the most probable when, after a comprehensive examination of both partners confirmed the absolute reproductive health of the man. The female factor is the cause of childlessness in 60% of cases. Female infertility is more often caused by functional disorders in the body, the treatment of which leads to a successful pregnancy and motherhood.

The most common causes of infertility are endocrine disorders in women. In this case, they speak of endocrine infertility.

The following hormonal disorders are distinguished:

  • Lack of ovulation;
  • Insufficient preparation of the endometrium for embryo implantation;
  • The death of the egg due to premature formation of the corpus luteum.

The second most frequent type of infertility is uterine. It is provoked by congenital and acquired defects in the structure of the uterus.
Tubal-peritoneal female infertility is diagnosed in the presence of defects and deformities of the fallopian tubes, as well as in the presence of adhesions in the pelvic area.
Specialists classify infertility into primary (if there has never been a pregnancy) and secondary (if there were births, abortions at any term, including ectopic pregnancy).

Why does female infertility develop?

Endocrine disorders of the process of ovulation and preparation for conception arise due to ovarian pathology (insufficiency, polycystic disease, etc.), disorders of the pituitary gland and thyroid gland.
Female infertility of uterine origin is caused by inflammatory processes in the uterus formed by synechiae (intrauterine adhesions), congenital defects in the development of the organ.

There are several known risk factors for infertility. These include:

  1. Age. Fertility decreases with age due to a decrease in the number of eggs and eventual depletion. Oocyte quality also deteriorates with age (especially after age 35).
  2. The inability to ovulate, i.e. anovulation, is usually due to an impaired secretion of LH and FSH by the pituitary gland. In polycystic ovary syndrome (PCOS), which manifests itself in the past or present, there is a decreased pulsation of GnRH released from the hypothalamus in case of an eating disorder, overweight or underweight or excessive exercise/low energy intake. This leads to an impaired release of FSH and LH from the pituitary gland. In hyperprolactinemia (high prolactin levels), LH/FSH secretion is affected, as well as a direct effect on the ovaries and endometrium (lining of the uterus).
  3. Tubal damage – Sexually transmitted infections (usually chlamydia) lead to inflammation and adhesions inside the fallopian tubes. Pelvic inflammation, such as appendicitis or surgery, can lead to adhesions that result in decreased motility and inability of the fallopian tubes to capture an egg when ovulating.
  4. Endometriosis – can affect fertility through inflammation leading to adhesions within the fallopian tubes, or between the fallopian tubes and other organs, cysts on the ovaries, or affect the endometrium and uterine contractility.
  5. Fibroids – depending on their size and location, can affect the uterine lining and uterine contractility.
  6. Testicular inflammation – seen as a result of post-pubertal mumps (can cause atrophy and absence of sperm (azoospermia) [25%] or reduced sperm count (oligospermia, in 50%).
  7. Epididymitis – sexually transmitted infections can cause scarring and vascular blockage.
  8. Radiation, chemotherapy, solvents and environmental toxins can also affect reproductive function and cause infertility.

The second most frequent type of infertility is uterine. It is provoked by congenital and acquired defects in the structure of the uterus.
Tubal-peritoneal female infertility is diagnosed in the presence of defects and deformities of the fallopian tubes, as well as in the presence of adhesions in the pelvic area.
Specialists classify infertility into primary (if there has never been a pregnancy) and secondary (if there were births, abortions at any term, including ectopic pregnancy).

The causes of tubal-peritoneal infertility lie in the violation of the patency of the fallopian tubes due to the formation of adhesions, deformities, complications of the inflammatory process. Tubal infertility is observed after removal of fallopian tubes or operations on them, and with adhesions of the pelvic organs – after any intervention.
Female infertility can be idiopathic – if the causes could not be identified, as well as autoimmune – if the partner’s sperm are killed in the cervical mucus due to the presence of anti-sperm antibodies.